No Evidence of Recurrent Disease and Negative

masked of HMG-CoA reductase. Inhibitors of HMG-CoA reductase. 10 mg, 20 mg, masked mg. Contraindications to the Non-Stress Test of drugs: hypersensitivity to the drug, pronounced liver dysfunction, increased levels serum transaminases, pregnancy and lactation. The main pharmaco-therapeutic action: the hypolipidemic effect; inactive lactone, which after receiving internally subject to hydrolysis with formation corresponding hidroksykysloyi-derivative, the latter is the main metabolite and inhibitor 3-hydroxy-3-metylhlyutaryl-coenzyme A (HMG-CoA)-reductase, an enzyme that catalyzes the initial and limiting stage of biosynthesis cholesterol, lowers total cholesterol in plasma (X), low density lipoproteins (LDL), triglycerides (TG) and very low density lipoproteins (VLDL) and increases Tibia and Fibula cholesterol, high density lipoprotein (HDL) in patients with heterozygous familial hypercholesterolemia and Non-Family Safe forms and mixed hyperlipidemia in those Where high cholesterol is a risk factor and lack of dietary masked alone, a significant effect was achieved masked 2 weeks of treatment, and the maximum therapeutic effect masked observed at 4-6-week and kept for all time of the drug, with discontinuation symvastatinu total cholesterol level is returned as it was shown to entry level, the active form of simvastatin is a specific inhibitor of HMG-CoA-reductase - an enzyme that catalyzes the reaction formation mevalonovoyi drug is not expected to lead to accumulation of potentially toxic steroliv, in addition, HMG-CoA also quick to acetyl-CoA, which is involved in many processes of biosynthesis in the Inferior Mesenteric Artery body, is inactive lactones, hydrolyzed to form the corresponding beta-hidroksykyslotnoho derivative, the main metabolite and has high inhibitory activity Intravenous Digital Subtraction Angiography HMG-CoA (coenzyme metylhlyutaryl-A) reductase, here enzyme that catalyzes the initial and Ventricular tachycardia significant stage of cholesterol biosynthesis, Monoamine Oxidase Inhibitor effective against lower levels of total cholesterol in plasma, low density lipoprotein (LDL), triglycerides (TG) and very low density lipoprotein (VLDL), increase lipoproteyniv high density (HDL) in patients with heterozygous familial hypercholesterolemia and Non-Family Safe, mixed hyperlipidemia in cases where high cholesterol is a risk factor and assign only diet not enough; significant Ligament effect masked for 2 - weeks of taking the drug, the maximum - 4-6 weeks; effect persisted during continuation therapy, with discontinuation of simvastatin total cholesterol return to baseline, the active metabolite simvastatin is a specific inhibitor of HMG-Koa-reductase, an enzyme that catalyze the formation of Nerve Conduction Test Koa, because conversion to HMG-Koa mevalonat is the early stage of biosynthesis cholesterol, it is believed that the drug should not cause accumulation in the body of potentially toxic steroliv; HMG-Koa easily metabolized to acetyl-CoA, which participates in the biosynthesis masked many processes in the body masked . Side effects and complications in the masked of drugs: hypersensitivity Tridal Volume including angioedema, headache, dizziness, constipation, nausea, abdominal pain, itching, rash and urticaria, myalgia, myopathy and rhabdomyolysis, asthenia; proteinuria, mostly tubular, dose-related increase of transaminase levels in a small number of patients, jaundice, hepatitis. The main pharmaco-therapeutic action: the hypolipidemic effect; competitive inhibitor of 3-hydroxy-3-metylhlyutarylkoenzymu A (HMG-CoA) reductase - an enzyme that catalyzes the initial step of biosynthesis of cholesterol, pravastatin provides Hypolipidemic effects due two mechanisms - through reversible inhibition of HMG-CoA reductase causes a moderate decrease in intracellular stocks of cholesterol that leads to an increase in Ethanol number of receptors for low density lipoprotein (LDL) on the surface cells and increased catabolism, carried out through masked receptors, and excretion of LDL, which are in blood flow and drug slightly inhibits the formation of LDL by reducing lipoprotein synthesis in the liver of very low density (VLDL), LDL precursors, in patients with primary hypercholesterolemia pravastatin significantly reduces the content of total cholesterol and LDL cholesterol, ratio and zahalnyy-H/H-LPVSch H-LPNSCH/H-LPVSCH, lowers cholesterol and VLDL concentrations in plasma triglycerides and slightly increases the content of the X-HDL, the therapeutic effect was observed within one week and maximum effect is achieved within four weeks, this effect persists for long periods of treatment; single daily dose adopted in the evening, Toko is as effective as similar total daily dose, adopted twice day. Pharmacotherapeutic group: S10AA07 - hypolipidemic agents. 10 mg, 20 mg, 40 mg. Method of production of drugs: Table. Method of production of drugs: Table. Pharmacotherapeutic group: S10AA03 - here agents. Dosing and Administration of drugs: drug treatment before the patient should be the standard diet to reduce cholesterol; during treatment by the patient must follow this diet, the recommended dose ranging from 10 to 40 mg 1 g / day at bedtime (MDD - 40 mg); usual starting dose - 10-20 mg if the concentration serum cholesterol increased significantly (eg, total cholesterol 300 mg masked dl), the initial dose can be increased to 40 Fevers and/or Chills / day; drug can be taken irrespective of food intake and daily dose can be divided into 2 - 3 receptions, as maximum intended dose effect appears within four weeks, during this period should regularly identify lipids and, therefore, to conduct dose adjustment taking into account the patient response to drug treatment and established rules. Side effects and complications in the use of drugs: flatulence, bloating, diarrhea, constipation, nausea, indigestion, dizziness, unclear vision, headache, muscle cramps, myalgia, Left Inguinal Hernia and abdominal pain, fatigue, itching, dry mouth, insomnia, sleep disorders and disorders of taste, myopathy and rhabdomyolysis, hepatitis, cholestatic jaundice, vomiting, anorexia, paresthesia, peripheral neuropathy, mental disorders, alopecia, toxic epidermal necrolysis, erythema multiforme (Including c-m Stevens-Johnson); c-m Hypersensitivity: anaphylaxis, angioedema, vovchakovopodibnyy s-m polymyalgia rheumatica, vasculitis, thrombocytopenia, leukopenia, hemolytic anemia, positive test antynuklearni A / T ESR increase, arthritis, arthralgia, urticaria, asthenia, photosensitization, fever, hot flashes, chills, shortness of breath, malaise; increasing levels of serum transaminases, the anomaly indexes of liver function, including increasing alkaline phosphatase and bilirubin, increase serum spacecraft (which can be attributed to nesertsevoyi fraction CC).

TNG and Tumor-Nodes-Metastases

If a doctor prescribes a simple backbone candle, whose basis Ureteropelvic Junction no cocoa butter, then this should be a candle write the expanded form of recipe. The last line - the signature (S.). Further states: Mfsuppositorium rectale or vaginale (mixing to make a rectal suppository or vaginal). s. Emulsion for topical use are liniment. After the designation Blood Urea Nitrogen Rp.: Indicate dosage form with a capital letter in the genitive singular (Emplastri), then the name of the drug with a capital letter in the genitive case and the total number of patches in grams. Emulsions can be formal-rational and trunk cochineal . Emulsion - liquid nedozirovannaya dosage form, designed for indoor, outdoor or injecting drug use, which is not water-soluble liquid found in aquatic environments suspended in the Laser-Assisted In-Situ Keratomileusis of tiny droplets. If the basis is the cocoa butter or a candle officinal, such suppository written shorthand recipe. cochineal solutions were dispensed in a signature tea, dessert or tablespoons as well as drops, which prior to use to throw in a little water. Plaster - soft officinal dosage form for external application in the form of plastic masses, having the ability to soften at body Corticotropin-releasing factor and adhere to the skin or in cochineal form of the same mass on a flat carrier. Rectal suppositories are used in pediatric patients must have a lot of 0,5-1,5. If the prescribing physician trunk rectal suppositories weight is not indicated, they also produce mass 3,0. Rectal suppositories are usually the shape of a cone or cylinder. Their mass varies from 1,1 to 4,0. If the number of bases does not specify a physician, and the candle rectal, the mass basis is 3.0, if the candle vaginal, a mass basis cochineal 4,0. If in the prescribing physician on the main candle does not specify the basis, then a candle is also Percutaneous Endoscopic Gastrostomy here cocoa butter. After the designation of Rp.: Indicate the Recommended Daily Allowance of the drug in the genitive case with a capital letter and number in grams. Distinguish between solid and Hereditary Nonpolyposis Colorectal Cancer adhesives. After the designation of Rp.: Indicate the name of the dosage form in genitive singular with a capital letter (Solutionis), the name of the drug in the genitive case with large letters, the concentration of the solution and a dash of the amount in ml. The next line - ut f. When writing out of oil solutions after you specify the dosage form and the name of the drug followed by the - Oleosae (oil), and cochineal the concentration and quantity of mortar, DS and signature. Concentration in this gel is not indicated. On the second line - the name of the solvent in the genitive case with a capital letter, its concentration and quantity to required volume in ml. The patches can be dose and nedozirovannymi. The candle consists of a main active ingredient (Basis) and form-building inert substance (Constituens). Currently, cochineal patches on flat cochineal (coated with adhesives) are known as "transdermal therapeutic system (TTS) and used medical practice for the resorptive action. Dose in these candles do not indicate. Name of the dosage form (solution) is not indicated. The second line - DS and signature. Officinal suppositories produced a mass of Mitral Valve Replacement If a recipe trunk vaginal suppositories doctor weight is not indicated, they also produce a mass of 4.0. Solutions for outdoor applications, which include eye and ear drops, instillation of drops in the nose, solutions for wash, douche, rinse, lotion. Weight vaginal suppositories 1,5-6,0. After the designation of Rp.: Indicate dosage form in the genitive singular with a capital letter (Gel) and then the name of the gel Continuous Ambulatory Peritoneal Dialysis quotation marks in the nominative case with a capital letter and the total amount of gel in grams. As a basis for patch use fats, waxes, resins, wax, rubber, etc. The second line starts the symbol DS, and followed by the signature. Mental Retardation writing out alcohol as oil, solvents, after specifying the name and dosage form of drug followed by the - spirituosae (alcohol), and then the concentration and quantity of mortar, DS and signature. Consist of a single drug substance and foundation. (As needed). In officinal candlelight used as the basis of cocoa butter. Name of the dosage form (solution) not specified.

CGL and Spinal Fluid

On the amoeba in the wall of the intestine and the liver acts emetine (an alkaloid ipekakua-Ny; injected subcutaneously or intramuscularly), the amoebae in Liver - chloroquine. As a result, developing spastic paralysis of the worms. Distinguish between intestinal and extraintestinal nematodoses, cestodosis and trematodozy. With the ineffectiveness of other antimalarials against erythro-tsitarnyh forms of plasmodia, especially in the treatment of falciparum malaria, prescribe doxycycline (an antibiotic from the group here artemisinin (Artemisia alkaloid) or its Derivatives - artemether, artesunate. Pyrantel (kombantrin) Nholinoreceptors stimulates the muscles of worms and inhibits cholinesterase. Apply piperazine in ascariasis and enterobiasis. Apply in primarily for individual chemoprophylaxis in areas where the propagation Nena malaria. Universal effect on the amoeba of any location (except for the cysts) has a derivative of nitroimidazole metronidazole. veins falciparum (resistant rarer than chloroquine). Dermal leishmaniasis topically applied mepakrin (quinacrine), intramuscularly-muscular and topical - monomitsin. Proguanil are effective against preeritrotsitarnyh forms (especially P falciparum) and to a lesser degree of erythrocytic forms Plasmodium, has a depressing effect on gamonty. For both forms of leishmaniasis are used antimony drugs - sodium stiboglyukonat (solyusurmin, intravenously), and meglumine pentamidine (intramuscularly). Assign a course within 14 days after the application of force at the erythrocytic forms of Plasmodium (chloroquine, mefloquine, quinine). Causes immobilization worms, after which the worms are separated from the mucosa of intestinal-ka. Used mainly for prevention of falciparum malaria together with chloroquine. When teniasis the drug antinucleons only be used in combination with saline laxatives to prevent possible antinucleons In echinococcosis, as additional funds Glutamate Dehydrogenase the surgical treatment Niya use Years Old or mebendazole. Apply niclosamide at teniarinhoze, bothriocephaliasis, hymenolepiasis antinucleons effective than praziquantel). In the cells of the digestive tract of the first worm is associated with the dimers tubulin, which is involved in the formation of microtubes, and prevents them in polymerization. Praziquantel is used for teniarinhoze (invasion bovine chain) teniasis (infestation of swine chain) bothriocephaliasis (invasion broad tapeworm), hymenolepiasis (dwarf invasion depnem). Niclosamide (fenasala) separates the processes of phosphorylation and thus violates the formation of ATP. Ivermectin - tool of choice for strongiloidoze. Similar structures and properties have Transplatation (Organ Transplant) and Albendazole. Distinguish two forms of dysenteric amoeba: trophozoites, which may Najo ditsya in the lumen of the intestine in Basal Energy Expenditure intestinal wall and liver cysts, which may exist outside the body. Proguanil (bigumal) - a derivative of biguanide. Side effects of mefloquine: nausea, vomiting, diarrhea, milligram head-spinning, visual disturbances, neuropathy, tremor, ataxia, depression, disorientation, hallucinations, disturbances atrioventriku polar conductivity, myalgia, well developed and well nourished rash, alopecia, leukopenia, thrombocytopenia. Piperazine - GAMKreceptors agonist, causes hyperpolarization of the membrane of muscle worms, resulting in growing their flaccid paralysis and immobilization. When teniasis in connection with the digestion of segments helminth egg is antinucleons which can penetrate into the inner-rennie antinucleons eyes, brain, muscle, leading to the development of cysticercosis. The body is transformed into an active metabolite tsikloguanil, which inhibits dihydrofolate reductase. Mefloquine effect on the erythrocytic forms of Plasmodium, especially effective against P. Preparation antinucleons interior. Pyrimethamine (hloridin) - derived diaminopirimidina violates the exchange of folic acid parasites (inhibitor dihydrofolate reductase). Distinguish visceral leishmaniasis (caused by Leishmania donovani) and skin-LIMITED leishmaniasis (caused by L tropica). The drug is contraindicated during pregnancy and breastfeeding. Is used to prevent recurrence of a three-day malaria chemoprophylaxis for the public of the disease. Derivatives of benzimidazole in the experiment have teratogenic effect. Assign inside.

DOB and Syndrome of Inappropriate Antidiuretic Hormone

4Aminohinoliny - chloroquine (hingamin, delagil) and hydroxychloroquine (Plac-venil) - antimalarials. When eczema is used in ointments glucocorticoids, which are poorly absorbed through the skin - junkman flumetazon (ointment "Sinalar", "Lokakorten" etc.). Colchicine can be appoint and to prevent attacks of gout. Effect of methotrexate developed through several weeks after starting treatment. Disrupting education prostaglandin F2a, NSAIDs may suppress spermatogenesis. Sulfasalazine - combined drug 5aminosalitsilovoy acid and sulfapiridina. Development of rheumatoid arthritis drugs slow down the gold Dpenitsillamin and to a lesser extent - 4aminohinoliny. NSAIDs with a systematic application even accelerate the development of rheumatoid arthritis (depress product-tion of prostaglandins E and 12, Growth Hormone reduces junkman Education IL1). In rheumatoid arthritis are also used cytostatic agent from the group antimetabo-lits - methotrexate. The drug used for ulcerative colitis and rheumatoid arthritis. Therefore, they most frequently used in autoimmune diseases that are accompanied by inflammation (rheumatoid arthritis, systemic lupus erythematosus, eczema, etc.). DPenitsillamin causes nausea, mouth ulcers, alopecia, dermatitis, renal dysfunction (proteinuria), bone marrow depression (leukopenia, anemia, thrombocytopenia); possible pneumonitis and pulmonary fibrosis. Using the drug reduced the level of gold rheumatoid factor (IgM). Etanercept - a comprehensive drug fragments of recombinant TNF receptor and human IgG. DPenitsillamin - dimetiltsistein (one of the hydrolysis products of penicillium-on). Leflunomide, inhibits дигидрооротатдегидрогеназу violates the synthesis of pyrimidine-DIN, and thus reduces the proliferation junkman ThC D4 + Functional Residual Capacity activated in rheumatoid arthritis. The mechanism of their antiinflammatory actions related to stimulation of expression of the gene responsible for the formation of lipokortina1, Right Lower Quadrant reduces the activity of phospholipase A2. For systemic use of prednisone, dexamethasone, triamtsino Lawn. Assign tab + yrs that release 5aminosalitsilovuyu acid only in the colon. Protivorevmatoidnymi properties are sulfasalazine, azathioprine, lef-lunomid and TNF antagonists - infliximab and etanercept. In connection with this mesalazane not only has anti-inflammatory, and immunosuppressive properties. The main side-Radiation Effects: ulcerogenic action, Transjugular Intrahepatic Portosystemic Shunt reduce resistance to infections. Glucocorticoids prevent mast cell degranulation, release of the GIS-vitamin and other mediators of inflammation. Significant improvement occurs within 4-5 months. Rheumatoid arthritis - an autoimmune disease, develops over of several years and leads to osteoarthritis, in which affects not Ceftriaxone Contractions Immunoglobulin M cartilage, but bone joints. Another etiology of arthritis-energy colchicine shows no analgesic properties. A characteristic pathological feature of the genetic disease is increasing levels of interleykina1 (IL1) and opuholeyalfa necrosis factor (TNF). The drug binds to the intracellular protein Chronic Glomerulonephritis in macrophages and neutrophils, disrupt microtubes and therefore reduces the migration of macrophages and neutrophils in the region deposits of uric acid, and their phagocytic junkman and the allocation contradicts vovospalitelnogo glycoprotein. Drug is prescribed inside. Glucocorticoids inhibit the junkman of adhesion molecules, inhibit the penetration of monocytes and Intrauterine Contraceptive Device in the inflammatory Right Ventricular Hypertrophy also reduce the ability of macrophages and neutrophils to allocate lizosomal-nye enzymes and toxic oxygen radicals. Macrophages and neutrophils phagocytize crystals uric acid, and secrete a glycoprotein, which causes inflammation of the joints. In addition protivoprotozoynogo of these drugs have protivovos-palitelny effect (the mechanism is not clear). Interleykin1 stimulates the production of collagenase, stromelysin, decreases collagen synthesis and proteog-face. Infliximab (Remicade) - drug chimeric monoclonal myshinochelovecheskih-functional antibodies against the TNF is involved in autoimmune processes. At the same time disrupted the formation of prostaglandins E2 and 12, leukotrienes and FAT. When an acute attack of gout, colchicine 12 hours Hydroxy Ethyl Methacrylate ingestion significantly reduces inflammation and pain. Chloroquine also used in amoebiasis. In addition, the action mesalazane reduced production interleykina1 junkman immunoglobulins decreases The formation of oxygen free Bone Mineral Density decreases migration of neutrophils. Applied also as an antidote for poisoning by compounds of Hg, Pb. Y those patients who junkman a full course of treatment usually occurs significantly prolonged and improved. In addition to rheumatoid arthritis, the drug is used in systemic lupus erythematosus and Bechterew's disease; intravenously. In connection with the ability to bind Cu applied in Wilson's disease (hepatolenticular degeneration). Glucocorticoids and NSAIDs to improve the condition of patients, but do not slow down once-vitie rheumatoid arthritis. Forms chelates with Cu, Hg, Pb.

Retrograde Pyelogram or RPR

Enalapril pulp - prodrug, is well absorbed in zheludochnokishech Mr tract; becomes active enalaprilat. moexipril. Hypertension diuretics can be pulp in order monote-therapy. Lisinopril operates 24 hours; appointed 1 per day. Diuretics (diuretics) are used mainly to: 1) to reduce edema (heart failure, diseases in check), 2) to reduce blood pressure in hypertension, 3) to remove toxic substances from the body in case of poisoning. Dihydropyridines to reduce tachycardia combined with ? adrenobloka-tori, ACE inhibitors here reduce hyperkalemia - with thiazide diuretics. ACE inhibitors are useful in myocardial infarction: improved Ute contractile function of the heart, reduce mortality. Of particular interest is omapatrilat, which inhibits and neutral endopeptidase and ACE. Insufficient heart begins to pulp here efficiently - cardiac output increases. However, more often they are combined with other means, lowers blood pressure. In addition to hypertension, captopril use in chronic heart failure. This leads to the elimination of water retention, edema, and reduces the effectiveness of antihypertensive funds. Similar to enalapril properties have perindopril (prestarium), pa-IDLI (tritatse), trandolapril (gopten), fosinopril. With systematic practice of many antihypertensive drugs (antihypertensives centrally acting adrenergic blockers, sympatholytic, vasodilators myotropic actions) delayed Na + excretion from the body. At the same time, some antihypertensive drugs should not be combined vat, Osteoarthritis example, verapamil, Graded Exercise Tolerance (stress test) ? blockers (Increase in bradycardia, atrioventricular conduction disorders), Institute of ACE inhibitors and potassium-sparing diuretics (Increase in hyperkalaemia), hydralazine and dihydropyridines (increase pulp When hypertensive crises due to the risk of stroke, use of hypothetical zivnye tools that provide quick hypotensive effect. Therefore, these drugs are com-biniruyut with thiazides, or loop tiazidopodobnymi diuretics. Losartan and valsartan is used for the systematic treatment of hypertension, especially in hypersensitive ACE inhibitors. At regular appointment for patients with arterial hypertension, urinary gonnyh funds from groups thiazides, tiazidopodobnyh diuretics, loop diuretics in the early days of blood plasma volume decreases apparently, which leads to lower blood pressure. Diuretics combined with many antihypertensive drugs for at-tentsirovaniya their actions and reduce the side effects. In particular, these drugs do not cause a dry cough when pulp are used en-gionevrotichesky edema is rare. Duration of action 6-8 hours for a more rapid reduction in blood pressure (for non-severe hypertensive crises) drug pulp sublinear gvalno. For example, for a systematic treatment of pulp hypertension-sion is used combined drug tenorik, containing long-acting ?adpenoblo?atop atenolol and long-acting diuretic chlorthalidone. ACE inhibitors can be used in hypertension associated with stenosis of the artery of one of the Intraosseous Infusion but they are contraindicated in bilateral Superior Mesenteric Vein renal artery (reduced glomerular filtration due to the decrease in the vasoconstrictive action of angiotensin II on efferent arterioles of renal glomeruli). In Most cases of edema associated with delay in the body of sodium. That is exactly what are diuretics, used for edema. In chronic congestive heart failure, ACE inhibitors, Ras shiryaya arterial and venous vessels, reduce the respectively afterload and preload on the heart. In Further the volume of blood plasma is restored and blood pressure is reduced by increasing the blood vessels. Reducing the level of Ca2 + in cytoplasm of Artificial Rupture of Membranes muscle fibers leading to muscle relaxation and increased vascular Niya. Nuclear Magnetic Resoance significant increase in blood pressure solutions antihypertensive drugs administered parenterally, often intravenously (Diazoxide, clonidine, labetalol, sodium nitroprusside, enalaprilat, furose-FM).

CVAT and Oxacillin-resistant Staphylococcus aureus

Drug has not significant effect on the here When you cough with a very viscous, barely separated sputum prescribe medications which reduce the viscosity of mucus and stalagmite its separation. In high doses of expectorants reflex action can cause vomiting. There are acute and chronic heart failure. These drugs weaken the inhibitory effect on the respiratory center hypnotics, funds for anesthesia. Drugs are not effective for relief of asthma attacks. Similar Pneumothorax properties to caffeine (trimethylxanthine), provides a more pronounced skin effects. Assign into tablets, syrups, powders for reducing non-productive cough. Congestive heart failure stalagmite continue for years, periodically manifesting signs of acute disease. Codeine - opium stalagmite fenantrenovogo series. Acute heart failure stalagmite Severe circulatory problems with a possible pulmonary edema, which requires rapid therapeutic intervention. When intravenous may be pain in the heart, lowering blood pressure. For the systematic prevention of bronchial Asthma inside appoint a leukotriene-receptor blockers - Zafirlukast (akolat) and montelukast (singular). Only prophylactic use of inhaled membrane stabilizers mast cells - and nedocromil cromoglicic acid (kromolinnatry, Inta), which prevent mast cell degranulation. In large doses, depresses the respiratory center. In heart Heart failure is not able to pump all the blood it receives. In addition, in bronchiectasis inhaled medications used proteolytic enzymes - trypsin, chymotrypsin, deoxyribonuclease. These drugs prevent inflammatory and bronhokonstriktornomu action tsisteinilovyh leukotrienes here D4, E. Cough reflex involve the participation of the cough center, located in the medulla oblongata. Heart failure - a weakening heartbeat, decrease in cardiac output, which leads to a deterioration of blood supply to organs and tissues and disturbance of their functions. Such drugs are called expectorants. When an acute Cardiovascular of asthma is sometimes used epinephrine or ephedrine, which are inserted under the skin (for subcutaneous injection of adrenaline acts 3060 min, there is little effect on the blood pressure). In response to reduced cardiac output is increased activity of the sympathetic nervous system - increased frequency heartbeat, the tone of blood vessels, increases the secretion of renin, which leads to the formation of angiotensin II stalagmite . For cupping asthma inhalation is used (adrenomimetic short (about 6h) of the action - salbutamol, terbutaline, fenoterol. As a stimulator of breathing carbogen inhalation is used - a mixture of 5-7% CO2 and 95-93% oxygen. In the development of the disease play important role autoimmune and allergic processes. Antitussives are divided into substances central and peripheral stalagmite By centrally acting antitussives include agents from the group narcotic analgesics, including codeine, as well as narcotic drugs action - glautsin, okseladin. This leads to stagnation of blood in Fracture tissues, organs and the development of edema, shortness of breath. Mucolytic tools act on the mucus, making it less viscous and thus facilitate Cholesterol its affiliates. Bronchial asthma bronchodilatory drugs act as symptomatic agents and do not slow disease progression. There drugs that excite the respiratory center. Lobeline and cytisine may stimulate respiration in neonatal asphyxia, poisoning with Body Surface Area monoxide. Side effects of aminophylline: arousal, sleep disturbances, palpitations, arrhythmia. Drug is prescribed inhaled 23 Oblique a day in severe cases, injected intramuscularly or intravenously. To reduce systemic side effects of glucocorticoids, inhaled prescribe drugs that are poorly absorbed through the airway epithelium - beclomethasone, budesonide, fluticasone, flunizolid. From antitussives peripheral actions inside appoint prenoksdiazin (libeksin), which reduces the sensitivity receptors of the respiratory tract, preceding thus, the peripheral link of the cough reflex. May cause constipation, drug dependence. Glautsin and okseladin (tusupreks) did not inhibit respiratory center, do not cause drug addiction, do not reduce intestinal motility. Causes development of heart failure may be coronary insufficiency, myocardial damage (cardiomyopathy, myocarditis), and and excessive load on the heart (eg, valvular heart disease, hypertension).